MRI findings. Transient epileptic amnesia <1hr, multiple attacks at time of presentation. Increased T2/FLAIR in hippocampus, thalamus and cortex . TIA/CVA. Amnesia in absence of other focal neurodeficits rare. DWI in vascular territories. Wernickes encephalopathy. More global amnesia and inattention
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DWI in vascular territories. Wernickes encephalopathy. More global amnesia and inattention The detection rate of diffusion‐weighted (DWI) hyperintense lesions varies widely in patients with transient global amnesia (TGA). The aim was to examine the association of hyperintense lesions on DWI magnetic resonance imaging (MRI) with patient characteristics, precipitating factors, clinical presentation and MRI settings in patients with TGA. Transient global amnesia (TGA) is a relatively uncommon neurological condition that is characterized by reversible disruption of short-term memory. In these patients, MRI of the brain often times reveal punctuate focal diffusion restriction lesion in mesiotemporal region that disappear without long term sequelae. Nakada T, Kwee IL, Fujii Y et al.
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Transient epileptic amnesia <1hr, multiple attacks at time of presentation. Increased T2/FLAIR in hippocampus, thalamus and cortex . TIA/CVA. Amnesia in absence of other focal neurodeficits rare. DWI in vascular territories. Wernickes encephalopathy. More global amnesia and inattention The detection rate of diffusion‐weighted (DWI) hyperintense lesions varies widely in patients with transient global amnesia (TGA).
We made a clinical diagnosis of transient global amnesia (TGA). Brain diffusion-weighted MRI at 48 h postevent showed focal bilateral medial temporal lobe high signal with restricted diffusion .
Transient global amnesia (TGA) is a temporary loss of short-term memory without other signs of impaired cognition and with absent focal neurological signs. The diagnostic criteria include that the attack should have been witnessed, there was no seizure or head injury, and the symptoms resolve within 24 hours.
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Diffusion-weighted MRI in transient global amnesia and its diagnostic implications. Kristina Szabo, Carolin Hoyer, Louis R. Caplan, Roland Grassl, Martin Griebe, Anne Ebert, View ORCID Profile. Michael Platten, Achim Gass. First published June 12, 2020, DOI: https://doi.org/10.1212/WNL.0000000000009783. Kristina Szabo.
Methods: MRI findings in 12 patients with clinical diagnosis of TGA were retrospectively analysed. 2018-07-27 · Transient global amnesia (TGA) has been a well-described phenomenon for more than 40 years. Clinically, it manifests with a paroxysmal, transient loss of memory function.
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2019-01-11 · Transient global amnesia (TGA) is a disorder characterised by a temporary, reversible disruption of short-term memory. While the diagnosis of TGA is based on its clinical features, neuroimaging is important to exclude other sinister causes of global amnesia. Furthermore, classical MRI changes in TGA have been well described in the literature. When transient global amnesia first occurs, MRI does not show specific abnormalities.
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Transient global amnesia is a disorder characterized by an attack of temporary anterograde amnesia with no other focal neurological symptoms. Recent studies showed that the characteristic MRI findings of this disorder are small punctuate diffusion restricted lesions in the CA1 area of the hippocampal formation.
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Visualisering, karakterisering och kvantifiering av hjärtinfarkt vid MRI av avlidna som ett alternativ till obduktion. Tre sfärer - En digital värld för kunskapsöverföring och global tänkande ´750813-0881 Amnesia´ av KTH Arkitektur, Kungl.
MRI brain was performed with a 3T scanner on 11 patients and 1 Transient global amnesia is a sudden, temporary episode of memory loss that can't be attributed to a more common neurological condition, such as epilepsy or stroke. During an episode of transient global amnesia, your recall of recent events simply vanishes, so you can't remember where you are or how you got there. Transient global amnesia is a disorder characterized by an attack of temporary anterograde amnesia with no other focal neurological symptoms. Recent studies showed that the characteristic MRI findings of this disorder are small punctuate diffusion restricted lesions in the CA1 area of the hippocampal formation.